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Editorial |

Clinical, Radiographic, Physiologic, and Biologic Measurements to Facilitate Personalized Medicine for ARDS

Michael A. Matthay, MD, FCCP; Jeremy R. Beitler, MD, MPH
Author and Funding Information

FINANCIAL/NONFINANCIAL DISCLOSURES: The authors have reported to CHEST the following: M. A. M. declares no conflict of interest for this editorial commentary and discloses support for his research from the National Institutes of Health/National Heart, Lung, and Blood Institute, Amgen, and GlaxoSmithKline. M. A. M. does consulting work for Cerus Therapeutics, GlaxoSmithKline, Boerhinger-Ingleheim, Bayer, Biogen, Quark Pharmaceuticals, and Incardia. None declared (J. R. B.)

aDepartments of Medicine and Anesthesia and the Cardiovascular Research Institute, University of California, San Francisco, San Francisco, CA

bDivision of Pulmonary and Critical Care Medicine, University of California, San Diego, San Diego, CA

CORRESPONDENCE TO: Michael A. Matthay, MD, FCCP, University of California, San Francisco, 505 Parnassus Ave, Room M-917, San Francisco, CA 94143


Copyright 2016, American College of Chest Physicians. All Rights Reserved.


Chest. 2016;150(5):989-990. doi:10.1016/j.chest.2016.05.013
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Diagnosis of ARDS requires that patients have a PaO2/FiO2 ≤ 300 mm Hg and bilateral infiltrates on chest radiograph that cannot be primarily attributed to cardiogenic edema or intravascular volume overload. ARDS, as clinically defined, includes patients with a wide range of underlying biologic processes. Identification of physiologic, clinical, and biologic characteristics that further classify patients with ARDS into more homogeneous subgroups or endotypes may add value in determining prognosis and predicting response to treatments.

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