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Diffuse Lung Disease: Connective Tissue-Associated Interstitial Lung Disease |

Correlation of Esophageal Dilatation and Pulmonary Fibrosis in Scleroderma

Vanessa Yap, MD; Massa Zantah, MD; Adarsha Selvachandran; Pardeep Athwal, MD; Electra Kaloudis, MD; Debapriya Datta, MD; Raymond Foley, MD
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University of Connecticut Health Center, Farmington, CT


Copyright 2016, American College of Chest Physicians. All Rights Reserved.


Chest. 2016;150(4_S):475A. doi:10.1016/j.chest.2016.08.489
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SESSION TITLE: Connective Tissue-Associated Interstitial Lung Disease

SESSION TYPE: Original Investigation Poster

PRESENTED ON: Wednesday, October 26, 2016 at 01:30 PM - 02:30 PM

PURPOSE: Pulmonary fibrosis has been reported to occur in 25-90% of patients with systemic scleroderma with a lower prevalence of 12-39% in limited scleroderma. The cause of this fibrosis is presumed to be post-inflammatory. However, because of the presence of esophageal dysmotility in scleroderma, there is a possibility of aspiration resulting in fibrosis. If this were the case, the worse the esophageal dilatation, the more extensive would be the fibrosis. The objective of our study was to determine if there is a correlation between esophageal dilatation (measured by the esophageal diameter and extent of pulmonary fibrosis (estimated by fibrosis score from high resolution CT [HRCT] of chest) in patients with scleroderma.

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