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Lung Cancer: Late-Breaking Abstracts: Lung Cancer Mechanisms |

RBM5 Inhibits the Proliferation of Cigarette Smoke-Induced Cancerous Transformation of BEAS-2B Cells

Zhang Jie, PhD; Lv Xuejiao, PhD; Hao Yuqiu, PhD; Su Zhenzhong, PhD; Meng Guangping, PhD; Wang Qi, PhD; Zhang Lin, PhD
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The Second Affiliated Hospital of Jilin University, Changchun, China


Copyright 2016, American College of Chest Physicians. All Rights Reserved.


Chest. 2016;149(4_S):A257. doi:10.1016/j.chest.2016.02.269
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SESSION TITLE: Late-Breaking Abstracts: Lung Cancer Mechanisms

SESSION TYPE: Original Investigation Slide

PRESENTED ON: Sunday, April 17, 2016 at 08:30 AM - 09:30 AM

PURPOSE: Cigarette smoking is the most important risk factor of lung cancer, and greater than 90% of lung cancers are cigarette smoke-related. Recent studies have indicated that the RNA-binding motif protein 5 (RBM5) has the ability to suppress tumor growth. The aim of this study was to investigate the role of RBM5 in cigarette-smoke extract (CSE)-induced cancerous transformation of bronchial epithelial cells and its regulation mechanism.

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