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Critical Care: Critical Care Nonpulmonary |

Enhanced Production of Nitric Oxide in A549 Cells Through Activation of TRPA1 Ion Channel by Cold Stress

Wenwu Sun, PhD; Zhonghua Wang, PhD; Zhuang Ma, MD; Haiyang Cui, PhD
Author and Funding Information

The General Hospital of Shenyang Military Region, Shenyang, China


Copyright 2016, American College of Chest Physicians. All Rights Reserved.


Chest. 2016;149(4_S):A172. doi:10.1016/j.chest.2016.02.178
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SESSION TITLE: Critical Care Nonpulmonary

SESSION TYPE: Original Investigation Poster

PRESENTED ON: Saturday, April 16, 2016 at 11:45 AM - 12:45 PM

PURPOSE: The respiratory epithelium is exposed to the external environment, and inhalation of cold air is common during the season of winter. In addition, the lung is a major source of nitric oxide (NO). In the present study, we investigated the effect of cold stress on the change of NO. Furthermore, we tried to explore the underlying mechanism of the change of NO using NO and Ca2+ signaling fluorescence imaging.

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