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Chest Infections: Chest Infections |

Interleukin-23 Facilitates Th1 and Th2 Cell Differentiation In Vitro Following Respiratory Syncytial Virus Infection

Jingjing Feng; Zhijun Jie, PhD
Author and Funding Information

The Fifth People's Hospital of Shanghai, Fudan University, Shanghai, China


Copyright 2016, American College of Chest Physicians. All Rights Reserved.


Chest. 2016;149(4_S):A81. doi:10.1016/j.chest.2016.02.086
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SESSION TITLE: Chest Infections

SESSION TYPE: Original Investigation Poster

PRESENTED ON: Saturday, April 16, 2016 at 11:45 AM - 12:45 PM

PURPOSE: Respiratory syncytial virus (RSV) infection induces activation and imbalance of the immune system; however, the role of T helper 17 cells (Th17) in the response to RSV infection remains unclear. Interleukin-23 (IL-23) is a key cytokine in Th17 cell differentiation. The aim of this study was to explore the function of IL-23 in determining the distribution of Th lymphocyte subsets (Th1, Th2, and Th17) after RSV infection in vitro.

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