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Allergy and Airway: Asthma III |

TLR2 Promotes Allergic Airway Inflammation Through JNK Signaling Pathway With Activation of Autophagy

Lei Fang; Rongyu Liu, PhD
Author and Funding Information

Institute of Respiratory Disease, The First Affiliated Hospital of Anhui Medical University, Hefei, China


Copyright 2016, American College of Chest Physicians. All Rights Reserved.


Chest. 2016;149(4_S):A28. doi:10.1016/j.chest.2016.02.030
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SESSION TITLE: Asthma III

SESSION TYPE: Original Investigation Poster

PRESENTED ON: Saturday, April 16, 2016 at 11:45 AM - 12:45 PM

PURPOSE: Toll-like receptors (TLRs) family as one of the best-characterized PRR families contributes to the pathogenesis of pulmonary diseases. Furthermore, autophagy such an immune process has been reported to be associated with asthma. Although numerous studies indicate that TLR2 activation led to contradictory effects in asthma, the role of TLR2 in OVA-induced mice model of asthma and the relationship with autophagy are little known.

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