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The Weighty Issue of Obesity Management in Sleep ApneaObesity and Sleep Apnea

Sanjay R. Patel, MD; Reena Mehra, MD, FCCP
Author and Funding Information

From Brigham and Women’s Hospital (Dr Patel), Harvard Medical School; and the Cleveland Clinic (Dr Mehra).

CORRESPONDENCE TO: Reena Mehra, MD, Cleveland Clinic, 9500 Euclid Ave, Cleveland, OH 44195; e-mail: mehrar@ccf.org


CONFLICT OF INTEREST: S. R. P. has received honoraria from the American Academy of Sleep Medicine, the American College of Physicians, and Elsevier Publishing; positive airway pressure machines and equipment from Philips Respironics; and grant funding from the ResMed Foundation, the American Sleep Medicine Foundation, and the National Institutes of Health. R. M. has received honoraria from the American Academy of Sleep Medicine, positive airway pressure machines and equipment for research from Philips Respironics, and funding from the National Institutes of Health.

Reproduction of this article is prohibited without written permission from the American College of Chest Physicians. See online for more details.


Chest. 2015;148(5):1127-1129. doi:10.1378/chest.15-1010
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Extract

The interrelationships between OSA and obesity are complex in large part because of the multidirectional nature of not only the association between these two factors, but also their relationships with undesirable cardiovascular health outcomes. Indisputably, obesity serves as a risk factor for OSA not only because of its direct mechanical effects (ie, disadvantageously affecting upper airways critical closing pressure), but also potentially via pathophysiologic pathways involving a reduction in lung volume resulting in a loss of caudal traction of the upper airways and upper airways neuromodulatory effects of adipokines.1

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