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Pulmonary Capillary HemangiomatosisPulmonary Capillary Hemangiomatosis: The Puzzle Takes Shape

David Langleben, MD
Author and Funding Information

From the Center for Pulmonary Vascular Disease, Division of Cardiology, Jewish General Hospital; and McGill University.

Correspondence to: David Langleben, MD, Room E-206, Jewish General Hospital, 3755 Cote Ste Catherine, Montreal, QC, H3T 1E2, Canada; e-mail: david.langleben@mcgill.ca


Financial/nonfinancial disclosures: The author has reported to CHEST that no potential conflicts of interest exist with any companies/organizations whose products or services may be discussed in this article.

Reproduction of this article is prohibited without written permission from the American College of Chest Physicians. See online for more details.


Chest. 2014;145(2):197-199. doi:10.1378/chest.13-2513
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In 1987, the late Dr Judah Folkman proposed the term “angiogenic diseases” for a group of disorders that seemed nonneoplastic but which involved persistent angiogenesis.1 This concept followed a rapid expansion in the identification and understanding of endothelial growth factors, vascular cell biology, and the essential role of the microvasculature in health and disease. It was recognized that the microvasculature seemed designed to remain mainly quiescent for many decades, with tight controls limiting capillary growth but with the ability to switch to rapid capillary proliferation in response to hormonal factors, inflammation and injury, or stimuli from tumors.1

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