Echocardiography-measured PASPs were available on 38 patients (63%) and in that subgroup correlated linearly with sPAP (r = 0.42, P = .009), mPAP (r = 0.33, P = .043), dPAP (r = 0.35, P = .03), and pulmonary vascular resistance (PVR) (r = 0.49, P = .002) (Fig 2, Table 3). In the 22 other patients, PASP was not measured because of the absence of a tricuspid regurgitant jet or an inadequate window. In a multiple linear regression model adjusting for age, race, sex, BMI, resting oxygen saturation, sleep apnea, congestive heart failure, and diabetes mellitus, the correlation between PASP and PVR remained significant (r = 0.34, P = .02) (Table 3), but the correlations between PASP and sPAP, dPAP, or mPAP were lost (Table 3). The correlation coefficients between PA:A ratio and echocardiography-measured PASP with sPAP were not statistically different, but the correlation of the PA:A ratio with mPAP was significantly stronger than the correlation between echocardiography-derived PASP and mPAP (z = 2.01, P < .05). In a multivariate logistic model, mPAP was independently associated with the presence of PA:A > 1 (OR, 1.44; 95% CI, 1.02-2.04; P = .04) adjusting for age, sex, race, BMI, resting oxygen saturation, sleep apnea, congestive heart failure, and diabetes (Table 4).