In cases of inaccurate pulse oximetry readings in the setting of nonpulsatile flow, intensivists must rely on other techniques to assess tissue oxygenation. We relied on ABG analysis with cooximetry. Other noninvasive means, such as transcutaneous cerebral oximetry, are starting to be used to assess Sao2 in settings of nonpulsatile flow. Like traditional pulse oximetry, cerebral oximetry is based on measurement of light absorption through tissue to estimate tissue oxygenation. But unlike pulse oximetry, cerebral oximetry assesses light absorption at the cerebral cortex, accounting for the effects of skin and skull absorption. Cerebral oximetry involves attaching various censors to the frontal head, and estimates Sao2 from venous blood (75%) and arterial (25%) blood samples. Data obtained from cerebral oximetry can be influenced by low cardiac output, hypotension, hypercapnia, hypothermia, bone absorption, and underlying cerebral pathology. Nevertheless, it has been shown to be useful in nonpulsatile flow.