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Exploring the Adipose Tissue-Lung Interaction in COPDAdipose Tissue-Lung Interaction in COPD

Emiel F. M. Wouters, MD, PhD, FCCP
Author and Funding Information

From the Department of Respiratory Medicine, Maastricht University Medical Center.

Correspondence to: Emiel F. M. Wouters, MD, PhD, FCCP, Department of Respiratory Medicine, Maastricht University Medical Center, PO Box 5800, 6202 AZ Maastricht, The Netherlands; e-mail: e.wouters@mumc.nl


Financial/nonfinancial disclosures: The author has reported to CHEST that no potential conflicts of interest exist with any companies/organizations whose products or services may be discussed in this article.

Reproduction of this article is prohibited without written permission from the American College of Chest Physicians. See online for more details.


Chest. 2012;142(4):820-821. doi:10.1378/chest.12-1030
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Extract

For many decades, COPD has been approached from a view of respiratory system involvement, and persistent and usually progressive airflow limitation associated with an enhanced chronic inflammatory response in the airways and the lung were characteristic findings. In the last decade, the definition of COPD has evolved to a disease with multicomponent involvement, and comorbidities, which are now assumed to contribute to the overall severity in individual patients.1 Instead of considering host-related factors to understand the heterogeneity of these comorbid conditions, the unifying concept of spillover of the local inflammation in the respiratory system was launched to understand the development and progression of this spectrum of comorbidities in COPD.

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