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Mechanism of Structural Remodeling in Pulmonary Fibrosis*

Yuh Fukuda, MD; Hiroshi Mochimaru, MD; Yasuhiro Terasaki, MD; Masashi Kawamoto, MD; Shoji Kudoh, MD
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*From the Department of Pathology and Internal Medicine, Nippon Medical School, Tokyo, Japan.

Correspondence to: Yuh Fukuda, MD, Department of Pathology, Nippon Medical School, 1–1-5 Sendagi, Bunkyo-ku, Tokyo 113-0022, Japan; e-mail: fukuda@nms.ac.jp



Chest. 2001;120(1_suppl):S41-S43. doi:10.1378/chest.120.1_suppl.S41
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Idiopathic interstitial pneumonia is pathologically classified into acute interstitial pneumonia (AIP), usual interstitial pneumonia (UIP), bronchiolitis obliterans organizing pneumonia (BOOP), nonspecific interstitial pneumonia (NSIP), and so on.1 UIP is clinically progressive and has a bad prognosis. AIP is a severe disease, but some patients recover with a suitable therapy. BOOP and NSIP have a generally good prognosis. We analyzed the mechanism of pulmonary structural remodeling in patients with AIP, UIP, BOOP, and NSIP, especially in the process and characteristics of early fibrotic lesions.

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