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Cellular and Molecular Pathobiology of Pulmonary Hypertension Conference Summary*

Marlene Rabinovitch, MD
Author and Funding Information

*From the Department of Pediatrics, Stanford University School of Medicine, Stanford, CA.

Correspondence to: Marlene Rabinovitch, MD, 269 Campus Dr, Room 2245B, Stanford, CA 94305-5162; e-mail: marlener@stanford.edu



Chest. 2005;128(6_suppl):642S-646S. doi:10.1378/chest.128.6_suppl.642S
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Extract

New knowledge concerning the pathobiology of pulmonary hypertension addressed cell-cell interactions, cell diversity and differentiation, and new attempts at understanding how mutations in a gene such as bone morphogenetic protein receptor (BMPR)-II cause aberrant intracellular signaling, and alter vascular cell structure and function. Common and complementary mechanisms were discussed that will lead to new and innovative hypotheses. Current scientific and therapeutic challenges were identified as well as new opportunities.

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