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Editorials |

Gut Mucosal Blood Flow : Regional Regulation or Systemic Pressure Dependence?

Weike Tao; Mali Mathru
Author and Funding Information

Affiliations: Dallas, TX
 ,  Galveston, TX
 ,  Dr. Tao is an Assistant Professor at The University of Texas Southwestern Medical Center. Dr. Mathru is the Clara Tibbetts Phillips Professor of Anesthesiology and the Director of the Surgical Intensive Care Unit, The University of Texas Medical Branch.

Correspondence to: Mali Mathru, MD, Department of Anesthesiology, The University of Texas Medical Branch, 301 University Blvd, Galveston, TX 77555-0591; e-mail: mamathru@utmb.edu



Chest. 2003;124(2):427-428. doi:10.1378/chest.124.2.427
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The intestines have been a major focus of investigations in critically ill patients, either as the “motor” or as the target organ of multiorgan dysfunction. Mucosal ischemia, and bacterial and endotoxin translocation have been implicated in a variety of situations such as shock, trauma, burn, sepsis, and cardiopulmonary bypass. It is generally believed that vasoactive substances and cytokines cause intense regional vasospasm and blood flow redistribution, leading to gut mucosal ischemia.1

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