remains an important cause of mortality on the ICU for which there are
no specific therapies. Factors predicting the onset or severity of this
syndrome are poorly understood, but the low incidence of ARDS in the
relatively large group of patients at risk of having this syndrome
develop suggests the involvement of genetic factors.1 No
specific genes have been identified to date. We have examined
genotype/allele frequencies for common polymorphisms in two candidate
genes: angiotensin converting enzyme (ACE) and interleukin (IL)-6.