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Genetic Polymorphisms Associated With Susceptibility and Outcome in ARDS*

Richard P. Marshall, MD, PhD; Suzanne Webb, BSc; Mike R. Hill, PhD; Steve E. Humphries, PhD; Geoffrey J. Laurent, PhD
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*From the Centers for Respiratory Research (Drs. Marshall, Webb, Hill, and Laurent) and Cardiovascular Genetics (Dr. Humphries), London, UK.

Correspondence to: Richard Marshall, MD, PhD, Centers for Respiratory Research and Cardiovascular Genetics, UCL, London, WC1E 6JJ, UK; e-mail: richard.marshall@ucl.ac.uk



Chest. 2002;121(3_suppl):68S-69S. doi:10.1378/chest.121.3_suppl.68S
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ARDS remains an important cause of mortality on the ICU for which there are no specific therapies. Factors predicting the onset or severity of this syndrome are poorly understood, but the low incidence of ARDS in the relatively large group of patients at risk of having this syndrome develop suggests the involvement of genetic factors.1 No specific genes have been identified to date. We have examined genotype/allele frequencies for common polymorphisms in two candidate genes: angiotensin converting enzyme (ACE) and interleukin (IL)-6.

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