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Editorials |

Is Asthma Another Interstitial Lung Disease?

Klaus Wassermann, MD, FCCP
Author and Funding Information

Affiliations: Cologne, Germany
 ,  Dr. Wassermann is Director of Pneumology and Bronchology, Medical Department III, University of Cologne

Correspondence to: Klaus Wassermann, MD, FCCP, Universität zu Köln, Klinik III für Innere Medizin, Kardiologie-Angiologie-Pneumologie und Internistische Intensivmedizin, Joseph-Stelzmann-Str. 9, 50931 Köln (Lindenthal), Germany; e-mail: klaus.wassermann@uni-koeln.de



Chest. 2002;121(3):673-674. doi:10.1378/chest.121.3.673
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Not quite so. But the description of inflammatory changes within the alveolar tissue13 along with the rediscovery of substantial defects in lung recoil4 may challenge the established “airways-only” dogma of asthma pathophysiology.5 We are used to equating low FEV1 in asthma patients with increased resistance due to bronchoconstriction and remodelled airway walls. The potential effect on airflow limitation of reduced parenchymal elastic force with a subsequent decline in expiratory driving pressure has been neglected. Actually, the relative contribution of either mechanism is of high interest.

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