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Mechanisms of COPD*: Conference Summary

Robert M. Senior, MD
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*From Pulmonary and Critical Care Medicine, Department of Medicine, Barnes-Jewish Hospital at Washington University Medical Center, St. Louis, MO.

Correspondence to: Robert M. Senior, MD, Pulmonary and Critical Care Medicine, Department of Medicine, Barnes-Jewish Hospital, 216 S. Kingshighway, St. Louis, MO 63110; e-mail: Seniorr@msnotes.wustl.edu



Chest. 2000;117(5_suppl_1):320S-323S. doi:10.1378/chest.117.5_suppl_1.320S-a
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Abbreviations: MMP = matrix metalloproteinase; VEGF = vascular endothelial growth factor

To begin, I want to thank Tom Petty and Steve Rennard for inviting me to be the Conference Summarizer. This is a great honor and a great challenge.

Having listened to the speakers and the discussions these past few days, I have two overriding impressions. The first is that research into the mechanisms of COPD is alive and well. The second is that the letter “C” in the acronym COPD might well stand for both “chronic” and “complex,” because it so evident that COPD has enormous complexity when one thinks about the pathophysiology at the cellular and molecular levels.

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