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Editorials |

A Light at the End of the Tunnel of Inflammation in Obstructive Airway Diseases?

Peter G. Gibson, MBBS
Author and Funding Information

Correspondence to: Peter G. Gibson, MBBS, Respiratory Medicine HMRI, Locked Bag #1, Hunter Mail Centre, NSW Australia 2310; e-mail: Peter.Gibson@hnehealth.nsw.gov.au

Professor Gibson is Director, Centre for Asthma and Respiratory Diseases, University of Newcastle and John Hunter Hospital, Newcastle NSW Australia.


The author has no conflict of interest to disclose.

Reproduction of this article is prohibited without written permission from the American College of Chest Physicians (www.chestjournal.org/misc/reprints.shtml).


Chest. 2008;134(3):475-476. doi:10.1378/chest.08-1478
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Extract

Chronic and persistent airway inflammation is one of the main driving forces in the obstructive airway diseases. It is proving increasingly useful to classify the pattern of inflammation based on the dominant granulocyte present, and there is great potential to use markers of inflammation serially to monitor disease activity and assist in treatment selection and dose adjustment. In asthma, this approach is well developed, particularly for eosinophilic forms of the disease. In this issue of CHEST (see page 628), Pallan et al1 start to bring some sense on how to bring a clinical approach to neutrophilic inflammation in asthma, particularly in the context of atypical infections.2

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